What is Amineptine?

What is Amineptine Medication?

Psychoactive Pharmaceuticals are over-the-counter or prescription drugs approved for human medicinal use. The inclusion of chemicals in this list is not intended to suggest that they are necessarily used recreationally. The substances described have a mind- or emotion-altering properties or may be listed because they have possible interactions with recreationally used chemicals.

Amineptine

Amineptine, formerly sold under the brand name Survector among others, is an atypical antidepressant of the tricyclic antidepressant family. It acts as a selective and mixed dopamine reuptake inhibitor and releasing agent, and to a lesser extent as a norepinephrine reuptake inhibitor.

Amineptine is an atypical tricyclic antidepressant and central nervous system stimulant. It is an indirect dopamine agonist, with additional stimulation of the adrenergic system. Its antidepressant effects are similar to other tricyclic antidepressants but it has a more rapid action, is better tolerated and has little cardiovascular, analgesic or anorectic effects.

In a double-blind, placebo-controlled study, the therapeutic efficacy of two antidepressants with different neurochemical mechanisms of action, amitriptyline, and amineptine, was investigated in patients affected by anxious depression. Sixty-six patients with the primary diagnosis of major depression or bipolar affective disorder (DSM-III-R) and meeting additional operational clinical criteria such as anxiety, trepidation, restlessness, early and/or late insomnia, impulsivity, hostility, dysphoria, compulsivity, hyper perspiration, palpitation, pollakiuria, and phobias were included. 

They were randomly assigned to three groups (n = 22) and treated either with placebo, amitriptyline (up to 100 mg/day) or amineptine (up to 200 mg/day) for 6 weeks. Patients showed a better response to amitriptyline, a preferential inhibitor of serotonin reuptake, than to amineptine, a selective inhibitor of dopamine reuptake. The present results suggest that alterations in serotonergic rather than dopaminergic transmission contribute to the pathophysiology of anxious depression.

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